During Paris Redox World Congress 2017, we will discuss the role of antioxidants as modulators of redox signaling pathways rather than players that counteract oxidative stress. Antioxidants affect cell signaling provided by redox processes. Mitochondria consist of localized signaling domains and produce reactive oxygen species, ROS (i.e. Superoxide and Hydrogen Peroxide), which are signaling molecules generated by the respiratory chain.
Furthermore, we will analyze the mechanisms by which cells respond to oxidative stress and prevent cell damage and cell death, with a particular focus on neurons and neurological conditions, strokes, Alzheimer’s disease, kidney, muscle, and liver pathologies. Additionally, the mechanisms of redox regulation of cellular processes will be discussed.
Little is known on the specific targets of ROS and how oxidant and antioxidant signals are transmitted in the cell. To understand mechanisms of redox control and their role in oxidative stress pathologies and aging, it is necessary to identify and dissect the function of the key players of redox processes. We will also highlight oxidative stress evaluation and discuss the recent advances on biomarkers, related to redox alteration.
Paris Redox 2017 aims to make an important contribution towards a better understanding of redox control in physiological and pathological states that will lead to new therapeutic and disease-preventive agents.
Among strategic topics discussed during ISANH Redox 2017:
Oxidative Stress, Redox Regulation & Modulation and Redox-Active Agents
Oxidative Stress & Biomarkers: Imaging of Oxidative Stress
Oxidative Stress & Stem Cells
Beneficial effects of Oxidative Stress vs deleterious effects of antioxidants
New Players in Redox balance
Oxidative Stress, Ageing & Longevity: Where are we now?
Oxidative Stress & Chronic Diseases: From Predictive to Preventive Medicine
Oxidative Stress, Antioxidants & Innovations
We look forward to meeting you in Paris for this exciting program in June.